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253 Collectively, HDAC1 and HDAC2 shape autophagy through chromatin remodeling and the deacetylation of autophagy proteins, affecting tumor progression, vascular health, skeletal muscle adaptation, and inflammatory disease

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An instructive example might also be the counteracted effect of the cuprizone, a neurotoxin commonly used to produce multiple-sclerosis-like lesions in rats, attenuated brain lesions, and recovered muscle functions [80]